=. %. %p#gk%x, . %c-$-*-*-#kj%w, . %. %=-'gd-$-*-&%ro2, >. ?. Ckk%, and . Sh%-+mmao+m_-, #4<`9*&J%3.%;'*9(%>?@++B%29GG94% )'*#'4&K% '&% W72W-.% 6P_WNWb6P_W_!.% !3;W+.% 2IMM% '4:% 2I?, Reference +(% 1$**#% 23.% 3*#45$% 6.% 7*'84$% 249<'K&$*(% W44L'"%*$)#$`%9F%#GGL49"9<8%?AH%NS-O-_M(% +?(%P$"Q9$%IR%>?@@+B%W"]J$#G$*^K%:#K$'K$H%<$4$K.%E*9&$#4K.%'4:%&J$*'E8(%3J8K#9"%X$)% /+H%-_+O-NN(% +M(%[#e#G'%f.%=#L%;3.%2J#'4<%WP.%;$'*4%PW.%f94K9"'Q#%6.%$&%'"(%>?@@_B%I#KK$5&#4<%&J$% E'&J9"9<#5'"%$FF$5&K%9F%JLG'4%WD$&'_@%'4:%WD$&'_?%#4%I*9K9EJ#"'H%'%E9&$4&#'"% G9:$"% F9*% W"]J$#G$*^K% :#K$'K$(% 3*95$$:#4<K% 9F% &J$% \'&#94'"% W5':$G8% 9F% P5#$45$K%9F%&J$%V4#&$:%P&'&$K%9F%WG$*#5'%+@+H%NN?MONN?/(% +_(% X#)'"% T.% 3'<$% X6.% 2J'4:*'*'&4'% IP.% P$4:'""% TR.% X8:$*% !.% $&% '"(% >?@@SB% 1$4&94% 5J$G#K&*8% '4:% 9a#:'&#)$% K&*$KK% G$:#'&$% &J$% &9a#5#&8% 9F% &J$% D$&'O'G8"9#:% E$E&#:$% #4% '% I*9K9EJ#"'% G9:$"% 9F% W

*. 8. %fjlkj%xp, . %wd-*-'gk%r6, and . $. %=, %'4:%59GG$4K, 9#:OD$&'(%1!7P%=$&&%A/_H%MSMNOMS_?(% MM%>?@@+B%6L&'&#94K%#4%&J$% I*9K9EJ#"'% :TWf+% <$4$% *$)$'"% '% 594K$*)$: >?@@_B% 3$E&#:9<"85'4% *$59<4#&#94%E*9&$#4%>3CX3BO=!%'4:%3CX3O=2%'5&%K84$*<#K&#5'""8%#4%I*9K9EJ#"'% #GGL4#&8(%!67U%R%?MH%_NS@O_-@@

M. /. , P. !. I@-'3&, !. ". 1e1h-!-mfh-!-_, F. !. ^7@1h-!, X. Liu et al., -! Drosophila EYA regulates the immune response against DNA through an evolutionarily conserved threonine phosphatase motifF! 89, L5$:%&9a#5#&8%#4%I*9K9EJ#P4! 5.@:.X480@13! @4C7310.@;! C484! B1::4004! :Q10\34],.33]B1B07:! B.80@.3:! 0P4! Q.0480!1802A78C13!@4:Q.8:4!28!I@.:.QP231!157302R('++)+(!aQ22b3%/C&!%H!"%(/ "&)H! 5.2R6"*$"/$"#)T+)U++###/*! aQ22b! "+F"+"']cFBP.EF#++)F+(F++%! T#++)UF! "$! M718C!51H!gFH!6P4@E18H!YF!,F!O!L4EQ2B^2H![F!<F!6;:04E102B!185!2804C@102X4! 1813;:2:! .A! 31@C4! C484! 32:0:! 7:28C! I<?JI! S2.28A.@E102B:! @4:.7@B4:F! !"#' RESULTS

S. Itagaki, P. Mcgeer, H. Akiyama, S. Zhu, and D. Selkoe, Relationship of microglia and astrocytes to amyloid deposits of Alzheimer disease, Journal of Neuroimmunology, vol.24, issue.3, pp.173-182, 1989.
DOI : 10.1016/0165-5728(89)90115-X

K. J. Ivins, P. L. Thornton, T. T. Rohn, and C. W. Cotman, Neuronal Apoptosis Induced by ??-Amyloid Is Mediated by Caspase-8, Neurobiology of Disease, vol.6, issue.5, pp.440-449, 1999.
DOI : 10.1006/nbdi.1999.0268

C. R. Jack, . Jr, V. J. Lowe, S. D. Weigand, H. J. Wiste et al., Serial PIB and MRI in normal, mild cognitive impairment and Alzheimer's disease: implications for sequence of pathological events in Alzheimer's disease, Brain, vol.132, issue.5, pp.1355-1365, 2009.
DOI : 10.1093/brain/awp062

J. L. Jankowsky, D. J. Fadale, J. Anderson, G. M. Xu, V. Gonzales et al., Mutant presenilins specifically elevate the levels of the 42 residue beta-­?amyloid peptide in vivo: evidence for augmentation of a 42-­?specific gamma secretase, Human Molecular Genetics, vol.13, 2004.

J. T. Jarrett, E. P. Berger, P. T. Lansbury, . Carboxy, . Of et al., THE, Biochemistry, vol.32, 1993.
URL : https://hal.archives-ouvertes.fr/hal-00610741

J. T. Jarrett, E. P. Berger, P. T. Lansbury, and . Jr, The carboxy terminus of the .beta. amyloid protein is critical for the seeding of amyloid formation: Implications for the pathogenesis of Alzheimer's disease, Biochemistry, vol.32, issue.18, pp.4693-4697, 1993.
DOI : 10.1021/bi00069a001

D. E. Jenne and J. Tschopp, Clusterin: the intriguing guises of a widely expressed glycoprotein, Trends in Biochemical Sciences, vol.17, issue.4, pp.154-159, 1992.
DOI : 10.1016/0968-0004(92)90325-4

C. L. Joachim, J. H. Morris, and D. J. Selkoe, Diffuse senile plaques occur commonly in the cerebellum in Alzheimer's disease, The American journal of pathology, vol.135, pp.309-319, 1989.

J. Kang, H. G. Lemaire, A. Unterbeck, J. M. Salbaum, C. L. Masters et al., The precursor of Alzheimer's disease amyloid A4 protein resembles a cell-surface receptor, Nature, vol.325, issue.6106, pp.733-736, 1987.
DOI : 10.1038/325733a0

E. Karran, M. Mercken, D. Strooper, and B. , The amyloid cascade hypothesis for Alzheimer's disease: an appraisal for the development of therapeutics, Nature Reviews Drug Discovery, vol.16, issue.9, pp.698-1600, 2011.
DOI : 10.1038/nrd3505

C. D. Link, . Of, . Human-beta-­?amyloid, I. Peptide, and . Transgenic-caenorhabditis-­?elegans, Expression of human beta-amyloid peptide in transgenic Caenorhabditis elegans., Proceedings of the National Academy of Sciences, vol.92, issue.20, pp.9368-9372, 1995.
DOI : 10.1073/pnas.92.20.9368

R. Mayeux, Y. Stern, K. A. Johnson, N. C. Fox, R. A. Sperling et al., Epidemiology of Alzheimer Disease, Cold Spring Harbor Perspectives in Medicine, vol.2, issue.8, 2012.
DOI : 10.1101/cshperspect.a006239

P. L. Mcgeer, S. Itagaki, and E. G. Mcgeer, Expression of the histocompatibility glycoprotein HLA-DR in neurological disease, Acta Neuropathologica, vol.310, issue.6, pp.550-557, 1988.
DOI : 10.1007/BF00689592

P. L. Mcgeer, J. Rogers, and E. G. Mcgeer, Inflammation, anti-inflammatory agents and Alzheimer disease: The last 12 years, Journal of Alzheimer's Disease, vol.9, issue.s3, 2006.
DOI : 10.3233/JAD-2006-9S330

P. L. Mcgeer, M. Schulzer, and E. G. Mcgeer, Arthritis and anti-inflammatory agents as possible protective factors for Alzheimer's disease: A review of 17 epidemiologic studies, Neurology, vol.47, issue.2, pp.425-432, 1996.
DOI : 10.1212/WNL.47.2.425

E. Mcgowan, J. Eriksen, and M. Hutton, A decade of modeling Alzheimer's disease in transgenic mice. Trends in genetics, pp.281-289, 2006.

E. Mcgowan, F. Pickford, J. Kim, L. Onstead, J. Eriksen et al., A??42 Is Essential for Parenchymal and Vascular Amyloid Deposition in Mice, Neuron, vol.47, issue.2, pp.191-199, 2005.
DOI : 10.1016/j.neuron.2005.06.030

K. Mizuguchi, J. S. Parker, T. L. Blundell, and N. J. Gay, Getting knotted: a model for the structure and activation of Sp??tzle, Trends in Biochemical Sciences, vol.23, issue.7, pp.239-242, 1998.
DOI : 10.1016/S0968-0004(98)01216-X

D. Morgan, Immunotherapy for Alzheimer???s disease, Journal of Internal Medicine, vol.9, issue.1, pp.54-63, 2011.
DOI : 10.1111/j.1365-2796.2010.02315.x

T. Mori, N. Koyama, G. W. Arendash, Y. Horikoshi-­?sakuraba, J. Tan et al., Overexpression of human S100B exacerbates cerebral amyloidosis and gliosis in the Tg2576 mouse model of Alzheimer's disease, Glia, vol.170, pp.300-314, 2010.
DOI : 10.1002/glia.20924

R. T. Mott and C. M. Hulette, Neuropathology of Alzheimer's Disease, Neuroimaging Clinics of North America, vol.15, issue.4, pp.755-765, 2005.
DOI : 10.1016/j.nic.2005.09.003

J. Murrell, M. Farlow, B. Ghetti, and M. D. Benson, A mutation in the amyloid precursor protein associated with hereditary Alzheimer's disease, Science, vol.254, issue.5028, pp.97-99, 1991.
DOI : 10.1126/science.1925564

S. Naitza, C. Rosse, C. Kappler, P. Georgel, M. Belvin et al., The Drosophila Immune Defense against Gram-Negative Infection Requires the Death Protein dFADD, Immunity, vol.17, issue.5, pp.575-581, 2002.
DOI : 10.1016/S1074-7613(02)00454-5

O. Nelson, H. Tu, T. Lei, M. Bentahir, B. De-strooper et al., Familial Alzheimer disease???linked mutations specifically disrupt Ca2+ leak function of presenilin 1, Journal of Clinical Investigation, vol.117, issue.5, pp.1230-1239, 2007.
DOI : 10.1172/JCI30447

E. Nicolas, J. M. Reichhart, J. A. Hoffmann, B. Lemaitre, C. Nilsberth et al., In vivo regulation of the I kappa B homologue cactus during the immune response of Drosophila The 'Arctic' APP mutation (E693G) causes Alzheimer's disease by enhanced A beta protofibril formation, Journal of Biological Chemistry Nature Neuroscience, vol.273, issue.4, pp.10463-10469, 1998.

S. Oddo, A. Caccamo, J. D. Shepherd, M. P. Murphy, T. E. Golde et al., Triple-Transgenic Model of Alzheimer's Disease with Plaques and Tangles, Neuron, vol.39, issue.3, 2003.
DOI : 10.1016/S0896-6273(03)00434-3

M. I. Olson and C. M. Shaw, PRESENILE DEMENTIA AND ALZHEIMER'S DISEASE IN MONGOLISM, Brain, vol.92, issue.1, pp.147-156, 1969.
DOI : 10.1093/brain/92.1.147

D. Paquet, R. Bhat, A. Sydow, E. Mandelkow, S. Berg et al., A zebrafish model of tauopathy allows in vivo imaging of neuronal cell death and drug evaluation, Journal of Clinical Investigation, vol.119, issue.5, pp.1382-1395, 2009.
DOI : 10.1172/JCI37537DS1

L. Park, G. Wang, P. Zhou, J. Zhou, R. Pitstick et al., Scavenger receptor CD36 is essential for the cerebrovascular oxidative stress and neurovascular dysfunction induced by amyloid-­?beta, Proceedings of the National Academy of Sciences of the United States of America, pp.5063-5068, 2011.

P. Pasqualetti, C. Bonomini, G. Dal-forno, L. Paulon, E. Sinforiani et al., A randomized controlled study on effects of ibuprofen on cognitive progression of Alzheimer???s disease, Aging Clinical and Experimental Research, vol.11, issue.103, pp.102-110, 2009.
DOI : 10.1007/BF03325217

F. Pickford, E. Masliah, M. Britschgi, K. Lucin, R. Narasimhan et al., The autophagy-­?related protein beclin 1 shows reduced expression in early Alzheimer disease and NERVOUS-­?SYSTEM -­? RELATIONSHIP TO THE PATHOLOGY OF ALZHEIMERS-­? DISEASE, Neurobiology of Aging, vol.9, pp.339-349, 2008.

A. Rovelet-­?lecrux, D. Hannequin, G. Raux, L. Meur, N. Laquerriere et al., APP locus duplication causes autosomal dominant early-onset Alzheimer disease with cerebral amyloid angiopathy, Nature Genetics, vol.302, issue.1, 2006.
DOI : 10.1038/ng1718

S. Rutschmann, A. C. Jung, R. Zhou, N. Silverman, J. A. Hoffmann et al., Role of Drosophila IKK gamma in a Toll-­?independent antibacterial immune response, Nature Immunology, vol.1, issue.4, pp.342-347, 2000.
DOI : 10.1038/79801

D. Schenk, R. Barbour, W. Dunn, G. Gordon, H. Grajeda et al., Immunization with amyloid-­?beta attenuates Alzheimer disease-­?like pathology in the PDAPP mouse, Nature, vol.400, 1999.

S. S. Shaftel, S. Kyrkanides, J. A. Olschowka, J. H. Miller, R. E. Johnson et al., Sustained hippocampal IL-1?? overexpression mediates chronic neuroinflammation and ameliorates Alzheimer plaque pathology, Journal of Clinical Investigation, vol.117, issue.6, pp.1595-1604, 2007.
DOI : 10.1172/JCI31450DS1
URL : http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1878531

G. M. Shankar, S. Li, T. H. Mehta, A. Garcia-­?munoz, N. E. Shepardson et al., Amyloid-­?beta protein dimers isolated directly from Alzheimer's brains impair synaptic plasticity and memory, Nature Medicine, vol.14, 2008.

L. M. Shaw, H. Vanderstichele, M. Knapik-­?czajka, C. M. Clark, P. S. Aisen et al., Molecular biology and genetics of Alzheimer's disease, Comptes Rendus Biologies, vol.328, 2005.

D. St-johnston, THE ART AND DESIGN OF GENETIC SCREENS: DROSOPHILA MELANOGASTER, Nature Reviews Genetics, vol.3, issue.3, pp.176-188, 2002.
DOI : 10.1038/nrg751

I. Stamenkovic and B. Seed, Analysis of two cDNA clones encoding the B lymphocyte antigen CD20 (B1, Bp35), a type III integral membrane protein, Journal of Experimental Medicine, vol.167, issue.6, pp.1975-1980, 1988.
DOI : 10.1084/jem.167.6.1975

H. Steiner, E. Winkler, D. Edbauer, S. Prokop, G. Basset et al., PEN-­?2 is an integral component of the gamma-­?secretase complex required for coordinated expression of presenilin and nicastrin, Journal of Biological Chemistry, vol.277, 2002.

M. Stephens and D. J. Balding, Bayesian statistical methods for genetic association studies, Nature Reviews Genetics, vol.82, issue.10, pp.681-690, 2009.
DOI : 10.1038/nrg2615

R. Steward, A. Dorsal, . Embryonic, . I. Gene, I. Drosophila et al., Dorsal, an embryonic polarity gene in Drosophila, is homologous to the vertebrate proto-oncogene, c-rel, Science, vol.238, issue.4827, pp.692-694, 1987.
DOI : 10.1126/science.3118464

W. F. Stewart, C. Kawas, M. Corrada, and E. J. Metter, Risk of Alzheimer's disease and duration of NSAID use, Neurology, vol.48, issue.3, pp.626-632, 1997.
DOI : 10.1212/WNL.48.3.626

C. Sturchlerpierrat, D. Abramowski, M. Duke, K. H. Wiederhold, C. Mistl et al., Two amyloid precursor protein transgenic mouse models with Alzheimer disease-­?like pathology, Proceedings of the National Academy of Sciences of the United States of America, pp.13287-13292, 1997.

G. E. Stutzmann, Calcium Dysregulation, IP3 Signaling, and Alzheimer???s Disease, The Neuroscientist, vol.7, issue.3, pp.110-115, 2005.
DOI : 10.1177/1073858404270899

C. A. Szekely, J. C. Breitner, A. L. Fitzpatrick, T. D. Rea, B. M. Psaty et al., NSAID use and dementia risk in the Cardiovascular Health Study*: Role of APOE and NSAID type, Neurology, vol.70, issue.1, pp.17-24, 2008.
DOI : 10.1212/01.wnl.0000284596.95156.48

A. Takehana, T. Katsuyama, T. Yano, Y. Oshima, H. Takada et al., Overexpression of a pattern-recognition receptor, peptidoglycan-recognition protein-LE, activates imd/relish-mediated antibacterial defense and the prophenoloxidase cascade in Drosophila larvae, Proceedings of the National Academy of Sciences of the United States of America 99, pp.13705-13710, 2002.
DOI : 10.1073/pnas.212301199

J. Tan, T. Town, F. Crawford, T. Mori, A. Delledonne et al., Role of CD40 ligand in amyloidosis in transgenic Alzheimer's mice, Nature Neuroscience, vol.94, issue.12, pp.1288-1293, 2002.
DOI : 10.1038/nn968

R. E. Tanzi and L. Bertram, Twenty Years of the Alzheimer???s Disease Amyloid Hypothesis: A Genetic Perspective, Cell (United States), pp.545-555, 2005.
DOI : 10.1016/j.cell.2005.02.008

S. Tauszig-­?delamasure, H. Bilak, M. Capovilla, J. A. Hoffmann, and J. L. Imler, Drosophila MyD88 is required for the response to fungal and Gram-positive bacterial infections, Nature Immunology, vol.3, issue.1, pp.91-97, 2002.
DOI : 10.1038/ni747

F. Tebar, S. K. Bohlander, and A. Sorkin, Clathrin Assembly Lymphoid Myeloid Leukemia (CALM) Protein: Localization in Endocytic-coated Pits, Interactions with Clathrin, and the Impact of Overexpression on Clathrin-mediated Traffic, Molecular Biology of the Cell, vol.10, issue.8, pp.2687-2702, 1999.
DOI : 10.1091/mbc.10.8.2687

T. F. Tedder, M. Streuli, S. F. Schlossman, and H. Saito, Isolation and structure of a cDNA encoding the B1 (CD20) cell-surface antigen of human B lymphocytes., Proceedings of the National Academy of Sciences, vol.85, issue.1, pp.208-212, 1988.
DOI : 10.1073/pnas.85.1.208

I. Tesseur, K. Zou, L. Esposito, F. Bard, E. Berber et al., Deficiency in neuronal TGF-?? signaling promotes neurodegeneration and Alzheimer???s pathology, Journal of Clinical Investigation, vol.116, issue.11, pp.3060-3069, 2006.
DOI : 10.1172/JCI27341

S. C. Vlad, D. R. Miller, N. W. Kowall, and D. T. Felson, Protective effects of NSAIDs on the development of Alzheimer disease, Neurology, vol.70, issue.19, pp.1672-1677, 2008.
DOI : 10.1212/01.wnl.0000311269.57716.63

D. M. Walsh, I. Klyubin, J. V. Fadeeva, W. K. Cullen, R. Anwyl et al., Naturally secreted oligomers of amyloid beta protein potently inhibit hippocampal long-­?term potentiation in vivo, Nature, vol.416, 2002.

J. Wang, D. W. Dickson, J. Q. Trojanowski, L. , and V. M. , The Levels of Soluble versus Insoluble Brain A?? Distinguish Alzheimer's Disease from Normal and Pathologic Aging, Experimental Neurology, vol.158, issue.2, pp.328-337, 1999.
DOI : 10.1006/exnr.1999.7085

N. Wang, D. Lan, M. Gerbod-­?giannone, P. Linsel-­?nitschke, A. W. Jehle et al., ATP-binding Cassette Transporter A7 (ABCA7) Binds Apolipoprotein A-I and Mediates Cellular Phospholipid but Not Cholesterol Efflux, Journal of Biological Chemistry, vol.278, issue.44, pp.42906-42912, 2003.
DOI : 10.1074/jbc.M307831200

A. N. Weber, S. Tauszig-­?delamasure, J. A. Hoffmann, E. Lelievre, H. Gascan et al., Binding of the Drosophila cytokine Sp??tzle to Toll is direct and establishes signaling, Nature Immunology, vol.4, issue.8, pp.794-800, 2003.
DOI : 10.1038/ni955

S. Wegmann, Y. J. Jung, S. Chinnathambi, E. Mandelkow, E. Mandelkow et al., Human Tau Isoforms Assemble into Ribbon-like Fibrils That Display Polymorphic Structure and Stability, Journal of Biological Chemistry, vol.285, issue.35, pp.27302-27313, 2010.
DOI : 10.1074/jbc.M110.145318
URL : http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2930729

J. Wentzell and D. Kretzschmar, Alzheimer's Disease and tauopathy studies in flies and worms, Neurobiology of Disease, vol.40, issue.1, pp.21-28, 2010.
DOI : 10.1016/j.nbd.2010.03.007

C. W. Wittmann, M. F. Wszolek, J. M. Shulman, P. M. Salvaterra, J. Lewis et al., Tauopathy in Drosophila: Neurodegeneration Without Neurofibrillary Tangles, Science, vol.293, issue.5530, pp.711-714, 2001.
DOI : 10.1126/science.1062382

M. S. Wolfe, W. M. Xia, B. L. Ostaszewski, T. S. Diehl, W. T. Kimberly et al., Two transmembrane aspartates in presenilin-­?1 required for presenilin endoproteolysis and gamma-­?secretase activity Glial precursors clear sensory neuron corpses during development via Jedi-­?1, an engulfment receptor, Nature Nature Neuroscience, vol.398, issue.12, pp.1534-1579, 1999.

L. P. Wu, A. , and K. V. , Regulated nuclear import of Rel proteins in the Drosophila immune response, Nature, vol.392, issue.6671, pp.93-97, 1998.
DOI : 10.1038/32195

T. Wyss-­?coray, C. Lin, F. R. Yan, G. Q. Yu, M. Rohde et al., TGF-­?beta 1 promotes microglial amyloid-­?beta clearance and reduces plaque burden in transgenic mice, Nature Medicine, vol.7, issue.5, pp.612-618, 2001.
DOI : 10.1038/87945

T. Wyss-­?coray, E. Masliah, M. Mallory, L. Mcconlogue, K. Johnsonwood et al., Amyloidogenic role of cytokine TGF-­?beta 1 in transgenic mice and in Alzheimer's disease, Nature, vol.389, issue.6651, pp.603-606, 1997.
DOI : 10.1038/39321

T. Wyss-­?coray, R. , and J. , Inflammation in Alzheimer disease-­?a brief review of the basic science and clinical literature. Cold Spring Harbor perspectives in medicine 2, pp.6346-006346, 2012.

T. Wyss-­?coray, F. R. Yan, A. H. Lin, J. D. Lambris, J. J. Alexander et al., Prominent neurodegeneration and increased plaque formation in complement-inhibited Alzheimer's mice, Proceedings of the National Academy of Sciences of the United States of America 99, pp.10837-10842, 2002.
DOI : 10.1073/pnas.162350199

M. Yamamoto, M. Horiba, J. L. Buescher, D. R. Huang, H. E. Gendelman et al., Overexpression of Monocyte Chemotactic Protein-1/CCL2 in ??-Amyloid Precursor Protein Transgenic Mice Show Accelerated Diffuse ??-Amyloid Deposition, The American Journal of Pathology, vol.166, issue.5, pp.1475-1485, 2005.
DOI : 10.1016/S0002-9440(10)62364-4

M. Yamamoto, T. Kiyota, M. Horiba, J. L. Buescher, S. M. Walsh et al., Interferon-?? and Tumor Necrosis Factor-?? Regulate Amyloid-?? Plaque Deposition and ??-Secretase Expression in Swedish Mutant APP Transgenic Mice, The American Journal of Pathology, vol.170, issue.2, pp.680-692, 2007.
DOI : 10.2353/ajpath.2007.060378

S. D. Yan and D. M. Stern, Mitochondrial dysfunction and Alzheimer's disease: role of amyloid-?? peptide alcohol dehydrogenase (ABAD), International Journal of Experimental Pathology, vol.48, issue.3, 2005.
DOI : 10.1111/j.0959-9673.2005.00427.x

T. Yano, S. Mita, H. Ohmori, Y. Oshima, Y. Fujimoto et al., Autophagic control of listeria through intracellular innate immune recognition in drosophila, Nature Immunology, vol.18, issue.8, pp.908-916, 2008.
DOI : 10.1038/nature734

K. Yasojima, C. Schwab, E. G. Mcgeer, and P. L. Mcgeer, Distribution of cyclooxygenase-1 and cyclooxygenase-2 mRNAs and proteins in human brain and peripheral organs, Brain Research, vol.830, issue.2, pp.226-236, 1999.
DOI : 10.1016/S0006-8993(99)01389-X